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By incorporating recent advances, this work provides a comprehensive understanding of the role of cytosolic multiprotein complexes, particularly inflammasomes, in the molecular pathogenesis of liver injury
Gut-flora-dependent metabolite trimethylamine-N-oxide promotes atherosclerosis-associated inflammation responses by indirect ROS stimulation and signaling involving AMPK and SIRT1
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Fourth, next-generation GBTs still undergoing clinical development were excluded from this analysis, preventing consideration of potential future advancements in the field