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The mammalian cytosolic thioredoxin reductase pathway acts via a membrane protein to reduce ER-localised proteins

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The compound reduces hematocrit and hemoglobin levels, probably by inducing oxidative stress in erythrocytes

These effects were achieved through direct inhibitory actions on prostatic GH-RH receptors, as evidenced by significant tissue-specific reductions in levels of NF-B/p50, cyclooxygenase-2, and androgen receptors, and changes in the expression of over 90 genes related to growth, inflammation, and signal transduction