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This strategy not only reduces aggregation risk but also enhances ADC circulation half-life and therapeutic performance

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Additionally, it indirectly reduces A2E photooxidation, lowers methylglyoxal levels, and inhibits the upregulation of the receptor for advanced end glycation products [245, 248]

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Unlike GSH, N-acetylcysteine NAC crosses the blood-brain barrier and might be expected to influence systemic and cerebral GSH levels

suggest that this peptide also interacts with ghrelin receptors located within the nervous system, potentially promoting signals associated with an observable elevation in hunger hormone signaling