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Treatment with the ferroptosis inhibitor Fer-1 reduced myocardial hypertrophy and pathological remodeling [237], underscoring the role of ferroptosis in hypertensive cardiac injury

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In a combined fertility and embryo-fetal development study in rats, subcutaneous doses of 0.01, 0.03 and 0.09 mg/kg/day (0.06-, 0.2-, and 0.6-fold the MRHD) were administered to male and female rats

A significant proportion is degraded before absorption occurs

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