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As an inhibitor of NRF2, Keap1 recognizes and binds to the ETGE and DLG motifs of NRF2 via its Kelch repeat domain, thereby continuously suppressing and promoting the degradation of NRF2 [87]

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After LPS stimulation, the expression of GLP-1R was significantly increased compared with that of the sham group (Fig

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CYP2E1 induction increases with repeated high-dose alcohol exposure and contributes significantly to ethanol clearance in individuals with alcohol use disorder (AUD)