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Published January 27, 2020 - More info Mechanisms mediating the cardioprotective actions of glucagon-like peptide 1 (GLP-1) were unknown

The exact mechanisms through which activation of THR improves MASH are still unclear but are known to control fat synthesis, regulate fatty acid oxidation, regulate cholesterol metabolism, improve mitochondrial function, and reduce inflammation and fibrosis

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Conversely, IGF-1 receptors are also widely expressed but exhibit higher densities in the cortex, hippocampus, and thalamus, with moderate expression in the olfactory bulb, hypothalamus, and cerebellum [95]

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