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These microbial and metabolic shifts collectively suppress NLRP3 inflammasome activation and downstream NF-B signaling in the CNS, resulting in attenuated neuroinflammation and enhanced cognitive performance

Both in this study and in the previous ones ( Table 1), the level of evidence did not allow to establish categorical recommendations, which can only be made with data from controlled clinical trials

Sulforaphane alone cannot directly stimulate autophagy at the 50 mg/kg dose associated with neuroprotection, but it could rescue rotenone-inhibited autophagy (Fig

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