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It was engineered to selectively activate the innate repair receptor (IRR) , a receptor complex involved in tissue protection and inflammation control

[8] The scientists suggest that GHRH-A [appeared to have] improved cardiac diastolic hemodynamics, including end-diastolic pressure, end-diastolic pressurevolume ratio, and stroke work, compared to placebo. Some laboratory studies reported a decrease in infarct size and attenuation of cardiac hypertrophy in mice