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The STEP and SURMOUNT trials consistently showed 26 to 40 percent of total weight lost was lean mass

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It is studied in research models for receptor binding, downstream signaling, and comparative pharmacology within the incretin receptor family

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In addition, our in vitro studies indicate a novel mechanism, that is GSTP can be phosphorylated at Ser184 and enter the nucleus under LPS stimulation, and GSTP interacted with HMGB1 to inhibit cPKC-mediated phosphorylation of HMGB1 and association between HMGB1 and CRM1 in the nucleus

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