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It does so by reducing melanin production through the inhibition of the enzyme tyrosinase, and by converting eumelanin (dark pigment) into pheomelanin (a lighter, reddish-yellow pigment)

These FFAs further activate inflammatory pathways, including nuclear factor-B and c-Jun N-terminal kinase, amplifying cytokine production and sustaining a self-perpetuating cycle of inflammation and metabolic dysfunction [19]

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