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The formation of this phenomenon is closely associated with the dysregulation of NF-B, matrix metalloproteinase (MMP), fibronectin (FN), -SMA, and connective tissue growth factor (CTGF), as well as the modulation of the renin-angiotensin-aldosterone system (RAAS) (Oatmen et al., 2020)

Cluster and pathway analyses were performed using ClusterProfiler on R

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