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The studies also had some shortcomings, as some only administered fluoxetine at the end of pregnancy and could not assess the effects of the drug throughout the entire pregnancy period, some did not explore the specific mechanisms of fluoxetine-induced fetal acidosis, hypocalcemia, and altered maternal estradiol metabolism, and some failed to assess the maternal and fetal health status over a long-term follow-up period

SK-MEL-103 displayed higher levels of ROS, accumulated more intracellular Fe 2+ and showed a moderate increase in levels of lipid oxidation (Fig