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In particular, accumulating evidence suggests that inflammation is a prominent etiology of OA, and that chondrocytes and synovial cells overproduce many of the soluble inflammatory mediators, such as cytokines, chemokines, growth factors, adipokines, prostaglandins, leukotrienes and regulators such as CPB [7, 8]

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This is used to determine the ability of the serum to stimulate IGF1R activity in vitro [3]

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Molecular Metabolism, 46 , 101102