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Part 2: pharmacotherapy

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2.2.3 Reactive oxygen species signaling While excessive ROS causes oxidative damage, physiological levels of mitochondrial ROS (mtROS) are crucial second messengers that activate key signaling pathways like NF-B and NLRP3 inflammasome, amplifying inflammatory responses and enhancing antimicrobial activity in phagocytes (40, 41)

It raises blood glucose levels by stimulating glycogenolysis and gluconeogenesis in the liver