While we observed no significant relationship between TA and CRP via linear regression, we did observe TA to be positively correlated with this non-specific marker of inflammation (\(\uprho\) = 0.29, p = 0.046)
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Due to the persistent NF-B activation signaling present within the TME and even normal tissues, the loss of functional p53 leads to increased NF-B-dependent inflammatory gene expression
There were faster granulation tissue formation, re-epithelialization, dermal remodeling, and collagen deposition through extracellular signalregulated kinases (ERK)1/2 signaling pathway as well as its downstream targets, including c-Fos, c-Jun, and egr-1
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