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How drugs stimulate T cells via pharmacological interaction with immune receptors

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A common feature of many of these neurotoxic compounds, such as rotenone, paraquat, or MPTP, is the inhibition of mitochondrial complex I, followed by the overproduction of ROS, ATP exhaustion, and induction of a wide range of abnormalities that can elicit neuronal and astrocytic cell death [54]

Moreover, effector T cells are capable of adapting their metabolism in response to nutrient limitation

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