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INTRODUCTION Oxidative stress results from an imbalance between reactive oxygen species production (ROS) and the abilities of biological systems to eliminate them

It was postulated that in response to the increased levels of intracellular zinc, vulnerable neurons would up-regulate ZnT-1, the plasma membrane-localized zinc transporter that facilitates zinc efflux (13)

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These findings correlated with elevated biomarkers of endothelial dysfunction (e.g., ICAM-1, VCAM-1) and pro - thrombotic cytokines [146]