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[] NAC in PTSD was studied with two more critical RCTs: one in Australia with 126 patients and 2.7 g/day, [] and another in the USA that uses 2.4 g/day to treat PTSD and concurrent alcohol dependence with a target of 200 patients

doi:10.1128/AEM.02173-12

Looking ahead, integrating precise diagnostics with tailored antioxidant therapies promises to transform the treatment of chronic disorders driven by OS, like DM2 and HTN

This may be the result of disturbed function of the ThDP-dependent 2-oxoglutarate dehydrogenase (Figure 1), as a universal effect of its inhibition is perturbation in relative amino acid abundance and a subsequent decrease in protein synthesis (196, 197)