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Excess extracellular glutamate inhibits and/or reverts the activity of the antiporter, thus depleting the cells of cysteine, a building block of the antioxidant glutathione, possibly increasing the cells vulnerability to oxidative stress [122]

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Specifically, it investigates the underlying molecular pathways through which UPF consumption may disrupt lipid metabolism

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It plays a key role in protecting cells from damage and supporting overall health

About this article Cite this article Boucelha, L., Djebbar, R