Administration of spironolactone, a selective MR antagonist, counteracted stress-induced mGlu2 suppression and the related development of depressive-like behavior, implicating rapid action of glucocorticoids via a receptor that is known to modulate glutamate release [20]
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It remains unclear why the addition of 5-amino-1MQ to exercise training produced a non-significant decline in muscle cell fats, but perhaps future studies attempting to replicate these findings will find significance if they include more mice in their experiments
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