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Artemisinin inhibits neuronal ferroptosis in Alzheimers disease models by targeting KEAP1

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Results Inhibition of glutathione metabolism depletes neuronal glutamate and affects excitatory transmission To test the hypothesis that glutathione is a significant reservoir for glutamate, we treated neuronal cells with molecular inhibitors targeting enzymes of the glutathione metabolic cycle: L-2-imidazolidone-4-carboxylate (2I4C), acivicin, L-buthionine sulfoximine (BSO), and sulforaphane

However, in this study we are not explicitly modeling how ROS is created from photo-oxidation

Finally, toxicity mediated by various types of drugs, such as bupivacaine [88], NSAIDs [89], clopidogrel [90], and even by lead and fluoride [91], as well as by NiCl 2 and KMnO 4 [92], was attenuated

The levels of glutathione depend on the mutation in question, and nrc1 , cad2-1 , pad2-1 and zir1 , which are affected in GSH1 show glutathione levels that range from 45 to 15% of normal levels 50,51,54,68