Wise LA, Radin RG, Palmer JR, Kumanyika SK, Boggs DA, Rosenberg L
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In activated macrophage cultures, GHK-Cu administration reduces the production of pro-inflammatory cytokines including interleukin-1, tumor necrosis factor-, and interleukin-6 by 40-65% while simultaneously increasing the production of anti-inflammatory mediators such as interleukin-10 and transforming growth factor-
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Given that obesity is a common comorbidity of inflammatory bowel disease (85), further research is needed to understand the significance of the GLP-1R downregulation during inflammatory bowel disease and how GLP-1 medicines interact with inflammatory bowel disease through both metabolic improvements and direct antiinflammatory actions on the intestine
Patients with: Personal or family history of Medullary thyroid cancer MEN2 syndrome Severe gastroparesis Pregnancy/ breastfeeding Pancreatitis or breastfeeding Severe depression Eating disorders Liver or kidney disease (caution advised) Why GLP-1 and Inflammation and Arthritis Matter We now understand that GLP-1 medications are not just weight loss drugs. They are anti-inflammatory, immune-modulating, metabolic medications that can dramatically shift health outcomes for patients with arthritis and autoimmune diseases
