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Rather than interacting with receptors on the plasma membrane of their target cells, the gasotransmitters (soluble gases such as nitric oxide and carbon monoxide, as unconventional neurotransmitters) can cross the cell membrane and act directly on molecules inside the cell [36]

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A 2018 review documented GHK-Cus modulation of over 4,000 human genes across tissue repair, antioxidant defense, DNA repair, and anti-inflammatory pathways (DOI: 10.3390/ijms19071987)

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Notably, redox signaling events are significant regulators of cell death pathways, among which GSH depletion has been demonstrated to be a vital early hallmark in the progression of different cell death mechanisms [18]