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Lifespan extension in genetically modified mice

This type of damage, often called cochlear synaptopathy, results from noise exposure causing a loss of synapses between cochlear hair cells and auditory nerve fibers, without affecting hair cell survival or audiometric thresholds 14

A landmark 2012 study found it influences the expression of over 4,000 human genes upregulating repair, collagen synthesis, and antioxidant defense while downregulating inflammation

[1] Fanelli S, Francioso A, Cavallaro RA, d'Erme M, Putignano P, Miraglia N, Mosca L

AMPK-mediated BECN1 phosphorylation promotes ferroptosis by directly blocking system Xc- activity

writingoriginal draft preparation, Z-ML