hypothesised that the obesogenic potential of TBT (0.05500 g/kg) could be sex-dependent, as proved by differential adipose and body weight changes, where male mice rapidly responded to low-dose TBT, translated by increased fat/body weight ratio and changed adipose tissue architecture [97], which may be related with the different androgenic milieu in male and female, as androgens are target regulators of lipid handling [98]
This pathway mitigates GPX4 ubiquitination and subsequent degradation in non-small cell lung cancer (NSCLC) cells (99)
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