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Furthermore, they confirmed that ZIP14 expression was enriched on the basolateral side and ZIP8 on the apical side (Figure 2)

C1A disrupts autophagic flux and induces cell death through substrate accumulation, 386 whereas other HDAC6 inhibitors increase the degradation of protein aggregates and increase immunity in glioblastoma

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doi: 10.1101/2023.08.04.23293636.

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