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2011 January 31 by Murat Oz Et al

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In vitro studies demonstrated its GH-releasing potency comparable to GHRP-6

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Fasting is required for CR-induced improvements in tau hyperphosphorylation and neuroinflammation, but not for CR-induced improvements in A plaque deposition To investigate the role of fasting in the ability of CR to attenuate AD pathology, we evaluated several pathological hallmarks of AD, including phosphorylation of tau, amyloid beta (A) plaque deposition, and gliosis