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We confirmed the effects of these two genes on cellular LD content by overexpressing SREBF1 and PLIN2
This underscores the necessity to develop new therapies targeting glutamatergic dysregulation to alleviate pain hypersensitivity without blocking glutamate receptors (Temmermand et al
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Increased ventricular wall thickness, chamber enlargement, enhanced stroke volume, and augmented diastolic filling represent adaptive responses to sustained training load and are generally associated with preserved or even supranormal functional reserve