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In addition, by reacting with oxidants such as superoxide anions, NO promotes cell injury and makes chondrocytes vulnerable to apoptosis induced by cytokines (Amin et al., 2000)

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In this study, using pro-atherogenic ApoE-deficient mice, liraglutide attenuated the development of atherosclerosis and improved plaque stability, as measured by a change in plaque composition, specifically an increase in collagen content and alfa-smooth muscle action

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