In translational research strategies evaluating antiarrhythmic concepts to be applied in patients with paroxysmal AF or following rhythm control by ablation or cardioversion, the use of cells obtained from subjects with persistent or even permanent AF exhibiting different cellular electrophysiological mechanisms constitutes a limitation that requires careful consideration
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It has gained significant attention in scientific research for its potential impact on key biological processes, including aging and cellular repair
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Furthermore, it reduces insulin secretion causing glucose dysfunction, enhances thermogenesis, adipogenesis and liver lipogenesis, limiting lipolysis at the same time (182)
