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The primary pathway is glucuronidation via UGT1A enzymes, which accounts for roughly 90% of clearance [1]
Integrating ChIP-seq, HiC, and PRO-seq datasets, we revealed broad functional binding of NRF2 at enhancer elements, including NRF2-dependent enhancer-derived RNA (eRNA) synthesis and extensive linkages between NRF2 target promoters and enhancers
Med Sci Monitor (2021) 27:e933446
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