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They significantly downregulated CHOP and GRP78 expression in diabetic mouse livers, implying a link between hepatic lipid dysregulation and endoplasmic reticulum stress

Salzillo, M

As discussed above, lipopolysaccharides (LPS) could prompt inflammation and amyloidogenesis in the brain, by inducing the disorder of TNF-a, IL-1, and cyclooxygenase-2 (COX-2) (87)

There is a distinct difference between feeling the medication and seeing the physical results

GLP-1 RAs exerted neuroprotective effects by inhibiting glutamate accumulation to prevent excitotoxicity and neural apoptosis [39], which possibly explains the protective effects of the GLP1R gene on background DR and severe nonproliferative DR, but not proliferative DR

12-Lipoxygenase metabolites of arachidonic acid mediate metabotropic glutamate receptor-dependent long-term depression at hippocampal CA3-CA1 synapses