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Nihon Kohden)

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Where estrogen works to restore the hormonal environment that governs fat distribution and metabolic regulation, GLP-1 therapy directly reduces caloric intake by recalibrating appetite signaling, improves glycemic control through multiple mechanisms, and drives the reduction of visceral fat mass that estrogen alone cannot reliably produce in the setting of significant metabolic burden

The GLP-1s were a big part of it

The effects of GLP1R agonists is not restricted to the brain, as it also has direct effects on the neurons that receive information from the gut (Vagal neurons) [21]