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For example, interleukin (IL)-6/STAT3 signaling is aberrantly activated in many CSCs, promoting self-renewal, immune evasion, and resistance to therapy, 63,64 whereas NF-B signaling supports CSC survival by sustaining inflammation-associated transcriptional programs

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They then exposed cells in blood vessels to it

[38] note that low adiponectin/leptin ratios exacerbate OS and inflammation

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