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The peptide is stable in gastric acid, which makes oral administration viable for gut-targeted applications, a property that distinguishes it from many other therapeutic peptides that degrade before reaching target tissue

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Back and co-workers [46], reported the catalytic cycle of di(3-hydroxy-propyl) selenide and acts an efficient catalyst for the reduction of t-BuOOH in the presence of BnSH

It activates two distinct receptor systems simultaneously: the GLP-1 receptor and the GIP receptor

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