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The FDA-approved drug diflunisal can also inhibit ACMSD, providing a structural base from which derivatives can be designed 237

Angiogenesis, Endothelium, and Blood Vessel Findings GHK-Cu reviews and wound-model studies discuss angiogenesis and blood vessel growth as part of tissue repair biology [10]

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Previous work has established that any residual OCR detected after oligomycin A treatment is due to non-respiratory oxygen consumption, which is usually ascribed to proton leakage through the mitochondrial inner membrane 79

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Inhibiting miR-3587 promotes upregulation of the HO-1 protein, which is encoded by HMOX1, and thus protects renal tissues from IR-induced ferroptosis [77]