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The glucagon receptor agonism, whilst counterintuitive given glucagon's role in raising blood glucose, is thought to increase energy expenditure and promote hepatic fat oxidation whilst reducing lipogenesis effects that may offer potential benefits in metabolic dysfunction-associated steatotic liver disease (MASLD)

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However, these technologies are years away from potential approval for human use

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Jamal et al

Glucose competence of the hepatoportal vein sensor requires the presence of an activated glucagon-like peptide-1 receptor