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Understanding the connection between MTHFR and longevity requires moving past the standard homocysteine narrative to the full mechanistic picture: how reduced methylation capacity accelerates the epigenetic aging clock, how it depletes the glutathione substrate pool, how it uncouples eNOS and accelerates vascular aging, how it impairs DNA repair through uracil misincorporation, and how it reduces the methylation-dependent gene regulation that FOXO3 and SIRT1 require to execute their longevity programs

Briefly, 100 age-matched male and female flies were segregated in separate vials and transferred to fresh vials during the experimental period every five days

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This is why we focus on liposomal deliverya process where the nutrient is wrapped in a phospholipid bilayer (a tiny bubble of fat) to protect it through the digestive tract