Toxicol Mech Methods (2003) 13(2):837
The damage caused by SAP to the vascular endothelium is manifested as the release of inflammatory mediators and leukocyte adhesion, increased vascular permeability, changes in blood flow shear stress, microthrombosis, and microcirculatory disorders
Los cidos grasos libres por accin de la Acil Coa sintetasa son convertidos a Acil CoA, que atraviesa libremente la membrana mitocondrial externa
To find out what else might be at play, we employed a mixture of public dataset analyses, in vitro and in vivo experimental models and discovered a potential driver: transcription factor NKX2-1 overexpression
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