By means of experimental models, it was found that in rat, RIP3/ (receptor-interacting kinase-3) diminishes inflammation, steatosis, and oxidative stress through the previously mentioned mechanisms and through the greater activation of NRF-2/HO-1 [56]
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Notably, differential susceptibility to ferroptosis exists between cell types: endometriotic cells acquire resistance through upregulation of GPx4 and GSH, whereas granulosa cells remain vulnerable due to decreased antioxidant defenses (35, 36)
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DHLA can donate electrons to these oxidized molecules, restoring them to their active, protective states
Misconduct in journals can lead to large numbers of retractions
