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The molecular mechanism by which the PfK13 mutations confer artemisinin resistance in clinical isolates may involve multiple cellular and metabolic processes, including hemoglobin uptake and digestion, the unfolded protein stress response, mitochondrial functions, oxidative stress responses (including tRNA thiouridine modification reprogramming), and an enhanced DNA damage repair mechanism 3, 811, 1320

Richards RS, Wang L, Jelinek H

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Oncoimmunology 10 , 1943253 (2021)

Storage & Stability Lyophilized -20C Stable for 24+ months when stored desiccated and protected from light Reconstituted 2-8C Use within 14 days

Potential new therapies ROS-based in CLL: an innovative paradigm in the induction of tumor cell apoptosis