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Under cystine starvation, the GCN2-eIF2-ATF4 pathway is activated, leading to an upregulation of CHAC1 expression

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For example, exposing endothelial cells to MOTS-c in vitro attenuated dysfunction caused by oxidative stress, in part by activating AMPK and suppressing inflammatory pathways (such as the MAPK/NF-B pathway) in those cells

To view a copy of this licence, visit About this article Cite this article Garca-Hidalgo, M.C., Mota-Martorell, N., Gonzlez, J

Oxygen-glucose deprivation in cell cultures kills all neurones within an hour, whereas astrocytes survive for several hours more