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investigated the influence of genetics on F 2 -isoprostanes, 8-oxo-7, 8-dihydro-2-deoxyguanosine, and 8-oxo-7, 8-dihydroguanosine (lipid-, DNA-, and RNA-derived oxidation products) in a population of elderly Danish twins [36]

Mouse model for molybdenum cofactor deficiency type B recapitulates the phenotype observed in molybdenum cofactor deficient patients

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12, 17 This triggers the release and/or production of neurotoxic and proapoptotic compounds such as nitric oxide synthase (NOS)

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