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Endoplasmic reticulum stress promoted acinar cell necroptosis in acute pancreatitis through cathepsin B-mediated AP-1 activation

Furthermore, reconstitution of wildtype (100) or LDL receptor deficient mice( Ldlr -/- ) (101) with Apoe -/- bone marrow accelerates atherosclerotic lesion development without affecting plasma cholesterol levels, demonstrating an atheroprotective role for macrophage apoE