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Moreover, HBV activated SIGLEC-3 on myeloid cells and induced immunosuppression by stimulating immunoreceptor tyrosine-based inhibitory motif phosphorylation and SHP-1/-2 recruitment via 2,6-biantennary sialoglycans on HBsAg

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p = 0.030), while the endothelial function of the GLP-1a group was maintained (2.1 0.6 vs