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Racial differences in serum selenium concentration: analysis of US population data from the Third National Health and Nutrition Examination Survey

As a result, ubiquitination and degradation of DHODH were inhibited, and the mitochondrial ferroptosis pathway was subsequently blocked [85]

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Notably, NO exhibits dual rolesphysiological concentrations modulate cerebral blood flow via cyclic guanosine monophosphate (cGMP) pathways, whereas pathological levels inhibit cytochrome c oxidase, impair mitochondrial respiration, and induce glutamate excitotoxicity through NMDA receptor S-nitrosylation [52]

However, more research is needed to confirm this effect in humans