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In these studies, at-risk alleles increased type I IFN response to environmental stress factors, whereas protective alleles reduced this signature (106, 107, 109, 110, 113115)

As individual peptide chains unfold, hydrophobic residues that were buried in the core become exposed to the aqueous environment

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Acyl-CoA synthetase long-chain family member 4 (ACSL4), an enzyme involved in the activation of PUFAs and facilitation of esterification into arachidonic acid (AA) and adrenoyl into PE, plays a crucial role in promoting ferroptosis [28, 29]

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