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In the initial inflammatory phase, IL-1 facilitates neutrophil infiltration and macrophage activation, whereas IL-18 supports angiogenesis and epithelial cell proliferation [30]

In contrast, when using smaller amounts of GspD that are present in whole cell lysates and visualized by immunoblot, neither the high-molecular-weight proteins nor the temperature-dependency were observed (compare Figs

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It is also known to be closely related to the T2MD metabolic syndrome, and the morbidity and mortality of CVDs (183, 184)

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