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Our findings highlight the beneficial effects of GLP-1 RAs in improving survival and reducing adverse cardiac and kidney events in individuals with type 2 diabetes and AKD

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While we did not conduct mechanistic pharmacological studies, we speculated and hypothesized, drawing on evidence from pharmacological research, that the pharmacological action of DPP4-i and their inability to cross the bloodbrain barrier offer a plausible explanation for differences in the risk of developing major and mild CI when compared to GLP-1a [6, 31, 32]

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