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The increase in cAMP levels in -cells is a consequence of GLP-1R activation in these cells, which in turn enhances the secretion of growth inhibitory hormone through signaling pathways such as PKA and Calcium/Calmodulin-dependent protein kinase (Brereton et al., 2015)

The standard protocol starts at 2.5 mg weekly with gradual escalation every four weeks based on response and tolerability, regardless of gallbladder status

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doi: 10.1016/j.amjmed.2017.04.043

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GLP-1 Receptor Activation Indirectly Reduces Hepatic Lipid Accumulation But Does Not Attenuate Development of Atherosclerosis in Diabetic Male ApoE- /- Mice